Can cooking with red palm oil reduce plaque buildup in arteries? or must I take a supplement?

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    3 Responses to “Can cooking with red palm oil reduce plaque buildup in arteries? or must I take a supplement?”

    1. Renie Says:

      Instead research magnesium because it is referred to as "nature’s calcium channel blocker." Here is an excerpt from

      It is widely believed that atherosclerosis (hardening of the arteries with fatty deposits) is an injury response. Meaning something is actually damaging the arteries, rather than just collecting stuff that is mechanically building up like silt in a river.

      Endothelial cells are a single layer of specialized cells that form the inside of your arteries. The subendothelial layer is a very thin connective tissue which contains elastin. As its name implies, this is the layer responsible for providing much of the elasticity in your arteries. It is a protein that is similar to collagen (an insoluble fibrous protein that is the primary component in connective tissue) and is a chief constituent in elastic fibers. Your body requires magnesium to maintain elastin.

      Smooth muscle cells are the next layer. Smooth muscle cells (SMC’s) provide integrity and control dilation of the arterial cavity. Endothelial cells respond to pressure by releasing agents that cause SMC’s to expand and contract. This controls blood pressure and flow in the artery. One of the earliest signs of magnesium deficiency is degeneration of the subendothelium. Animals with low magnesium diets have been shown to lose the elasticity of their arterial system. Coronary arteries require more elasticity than other arteries because the heart expands and contracts as it beats. Since these arteries are on the heart muscle itself, they too must stretch and flex as the heart beats. Continuing loss of elasticity results in inflammation of the endothelial and subendothelial layers at points that are most mechanically challenged by stretching. Imagine a small rubber-band like tube shaped to form the letter "Y". In your mind’s eye grasp the two legs of the Y in one hand. With your other hand, grip the single leg. Begin pulling them apart just as though they were stretching on the surface of the heart. Stretch it as far as you can. Where is the shape weakest? If we left the rubber tube out in the sun for a week or so, what would happen if you slowly stretched it again? Where might you expect the first crack to appear? Maybe not always, but most of the time I believe it would happen at or near where one tube becomes two… at the bifurcation. If your artery loses elasticity does it make sense that the problem might show up at or near the bifurcation?

      Lipoproteins are fats that are combined with proteins comprising cholesterol. Although not completely accurate, think of lipoproteins as little sausages. They have a casing and inside is the cholesterol. There are essentially two kinds of lipoproteins (sausages); high density lipoprotein (HDL) and low density lipoprotein (LDL). HDL is considered the good cholesterol while LDL has been linked to heart disease. (VLDL is another class of lipoprotein known as very low density lipoprotein. It’s the really bad LDL.)

      When inflamed, endothelium release cytokines (cellular hormones) that can, like lipoproteins, have good or bad effects on the artery. One type of cytokine constricts arteries and increases coagulation; another dilates arteries and protects against clumping of platelets on the endothelium. Cytokines chemotactic (chemically activated) for monocytes (specialized white blood cells), provide adhesion sites at the inflammation. Monocytes burrow beneath the enodothelial monolayer, ingest lipoproteins, and secrete agents to stop the inflammation.

      It has been demonstrated that monocytes engorged with oxidized low density lipoprotein (LDL) over-ingest. (They don’t know when to stop eating the low density lipoprotein.) The term is they are not "upward regulated", and therefore these cells burst into foam cells. (They pig out on LDL sausages and rupture their little membranes.) When this happens, they release free cholesterol, cytokines, and procoagulants (thrombosis inducing agents) into the surrounding area. This process forms plaque which consists of a mass of lipid-engorged monocytes covered by a fibrous cap. The whole ugly mess is pushed into the artery by the smooth muscle cells. Injury to the endothelium is the first pathogenic step, or genesis of atherosclerosis, therefore it is called atherogenesis. (Literally "gruel origin"). This gruel is usually referred to as arterial plaque. However, even before the plaque begins, lesions are formed in the endothelium. The medical world does not yet clearly understand what causes these lesions. If they did, they might be able to prevent atherosclerosis and maybe even arteriosclerosis. Since LDL ruptures monocytes when they "eat" too much oxidized lipoprotein (LDL), lessening the LDL/HDL ratio plays a role in lowering your risk of heart attack. (A ratio higher than about 3.6 LDL/HDL is considered high). Some believe that LDL/HDL ratio may be more important than your cholesterol level.

    2. SkepDoc 3.0 Says:

      There is no evidence for this..the opposite in fact. Palm oils in general…and red palm oil is no exception…are one of the more unhealthy vegetable oils, and their use is associated with INCREASED levels of triglyceride and LDL (aka bad cholesterol)

    3. ƦєdAиgєℓ Says:

      Yes, renie is right, plaque build up is an injury response but it is known what causes the damage (there is just no pharmaceutical drug to lower it! hence no money..vitamin B6, B12 and folate all lower high homocysteine levels…i’m getting ahead of myself lol) so what is the damage caused by?…an amino acid in the blood called homocysteine. When this amino acid is too high in the blood, it changes LDL cholesterol to damaging LDL which severely attack the artery walls. It’s not the amount of cholesterol, although you don’t want to eat too much fat anyway, (50% of people who have a heart attack have normal cholesterol levels), it’s the homocysteine that changes the cholesterol you already have in the blood to dangerous cholesterol. Think of it as slivers of glass running through your veins all through your body and brain. Cholesterol does it’s job at patching up the damage and the immune sends out macrophages to help with the damage control. High homocysteine levels cause macrophages to become more sticky causing even more thicker deposits on the arteries and eventually a blood clot cannot get through (high homocysteine levels causes the blood to clot easier)..bang heart attack or stroke.

      Homocysteine level should be around 6.3 umol/L for good health. A good supplement is systematic enzymes. The are the garbage collecters of enzymes and listed as one of it’s jobs is clearing up plaque buildup. I’m all for that! 🙂 Studies show that adding palm oil into the diet can remove plaque build up in arteries as well so yes to your question, but taking systematic enzymes can only help with the process. But good old B vitamins are a winner to prevent a toxic build up of homocysteine. Have a blood test and see what level you are. 🙂

      Homocysteine: (page 26 goes into how homocysteine damages the arteries)

      Systematic enzymes:

      Red Palm Oil: